Results support the hypothesis of cognitive reserve in traumatic brain injury
Kessler Foundation researchers have found that higher educational attainment (a proxy of intellectual enrichment) attenuates the negative impact of traumatic brain injury (TBI) on cognitive status.
Single protein controls Drosophila nervous system development and survival
This research could also potentially impact how science and healthcare think about and treat brain injuries, Kuo said. Currently, damaged neurons that have lost their dendrites are unable to properly communicate with their neighbors, rendering them nonfunctional. The problem could be reversed, he said, by helping neurons modify their original developmental program and regrow new dendrites.
“Think about it: the brain is built for logic, so its organization must be logical.”
Researchers have identified eight distinct neural subnetworks that together form the connectivity infrastructure of the mammalian cortex, the part of the brain involved in higher-order functions such as cognition, emotion and consciousness. This study is the first comprehensive mapping of the most developed region of the mammalian brain: the cerebral cortex. The cortex is highly complex and made up of many densely interconnected structures, but when you strip it down, is organized into a small number of subnetworks
“With the brain,” Dr. Reid said, “you can always go deeper.”
There are many ways to map the brain and many kinds of brains to map. Although the ultimate goal of most neuroscience is understanding how human brains work, many kinds of research can’t be done on human beings, and the brains of mice and even flies share common processes with human brains.
Taking acetaminophen during pregnancy is associated with a higher risk in children of attention-deficit/hyperactivity disorder and hyperkinetic disorder.
Acetaminophen can cross the placental barrier and it is plausible that acetaminophen may interrupt fetal brain development by interfering with maternal hormones or through neurotoxicity, such as the induction of oxidative stress, which can cause the death of neurons.